OldRegular
Well-Known Member
There is an appropriate saying that was once prevelant among computer people and seems appropriate for the subject of this thread: Crap in, crap out!
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Ok.Maybe you could go through a few examples of actual increases in information and how you would measure this increase. Then maybe you could take a generic example of a duplication and mutation which results in a new gene with a new function and tell us why this is not new information. You have been given several examples of such and a literature search would generate thousands of additional examples.
But what about mutation? A simple random mutation, while again fulfilling Shannon information, will not meet Gitt's new version of information.
But if that mutation makes a new gene which codes for something useful, things change. We still have the underlying syntax of the genetic code, so the semantic and pragmatic level are undeniably met. But since the mutated version is useful, it now has a purpose and meets the reqirement of the apobetic level as well. Once the gene mutates into something useful, then its purpose cannot be differentiated in any way from the purpose of any other functional gene. The requirements for even Gitt have been met by this process. New information has been created in a manner consistent with evolution's requirements.
While quantifiable is not a normal word in my vocabulary, let me try to address what you are saying.The question is if you have a given genome or part of one and if a mutation (or mutations) can you give us a quantifiable measure of whether new "information" has been provided or not?
This is an interesting statement. You have no observational evidence that the fossil record contains these transsitional forms (ie no one was there to see it happening) - only the discreet "here is one with gills... and here is one that had evolved lungs". Yet nothing in between can ever be found. You yourself state that transsitions would be long and complex, yet this isn't at all what the fossil record represents. We see discreet creature after discreet creature with no observational evidence whatsoever that one transformed into another. We don't ever see transsisions with part of a system - it goes rapidly from one discreet fully functioning system in one animal to another animal with a completely different fully developed, functioning system. This indicates that (if one believes in evolution) evolution happens very, very fast and the transsitional creatures are very short lived - so short they don't show up in the fossil record. In this case, we should be able to observe these changes in real time currently. But we do not. We don't see additions of information (as in our book analogy). We do see corruptions, losses, and re-arranging, however. This is consistent with all the other current observations we can make (such as entropy and thermodynamics). Our current (and actual) observations fit much better with a perfect creation which has been de-evolving ever since than with an imperfect creation that has been evolving ever since. This, of course, is much more in line with scripture than the theory of evolution.These are not examples of single mutations. These are long, complex processes. Of course we can also follow where most of them actualy happened in the fossils record.
"Yet nothing in between can ever be found. You yourself state that transsitions would be long and complex, yet this isn't at all what the fossil record represents. We see discreet creature after discreet creature with no observational evidence whatsoever that one transformed into another. We don't ever see transsisions with part of a system - it goes rapidly from one discreet fully functioning system in one animal to another animal with a completely different fully developed, functioning system."But what about mutation? A simple random mutation, while again fulfilling Shannon information, will not meet Gitt's new version of information.
But if that mutation makes a new gene which codes for something useful, things change. We still have the underlying syntax of the genetic code, so the semantic and pragmatic level are undeniably met. But since the mutated version is useful, it now has a purpose and meets the reqirement of the apobetic level as well. Once the gene mutates into something useful, then its purpose cannot be differentiated in any way from the purpose of any other functional gene. The requirements for even Gitt have been met by this process. New information has been created in a manner consistent with evolution's requirements.
The question to be asked is what interpretation best explains the evidence that we observe. As new information comes in, it will either support your interpretation or contradict it. Support tells you that you are on the right track. Contradiction tells you that you need to rethink your theory.
The first observation that gets discussed is morphology. Now if we look at the animals that are alive today and the animals that the fossil record tells us were alive in the past, we see that the form a nested heirarchy. All by itself this could mean that all of these animals were produced by common descent or by common designer. (As a note, I am not trying to set up a false dilemma here. I recognize that there may be other explanations that could be put forward but I am purposely restricting the discussion to the two possibilities under discussion.) So you have to go to the next observation.
The next most obvious observation is genetic. If you examine all the different types of genetic material that has been tested and use it to construct phylogenic trees, you find that you get much the same pattern as you do when you arrange the fossils by morphology. Let's see how these observations stack up.
One easy test is to look at just the functional genes. These can again be used to support common descent or a common designer. Both will claim that creatures that are the most similar should have the most similar DNA.
But you can start to untangle the two by looking into further types of genetic material that is not related to the functional part of DNA. One example would be to look at retroviral inserts. These happen when a virus inserts part of its genome into its host. If this happens in a reproductive cell, then the genome of the virus can be passed on to the offspring. Since this has nothing to do with the functional part of the genome, it can shed light on the situation for us. For example, if common descent were true, then you would expect the retroviral DNA to show the same pattern as the other lines of observation. If a common designer were the true explanation, then you would expect a random distribution of the retroviral inserts when compared between the species. In fact, you see that the pattern follws that which wbe expected of common descent. The common designer option cannot explain this pattern.
If you take the retroviral discussion and repeat it with things like paralogs, pseudogenes, retrotransposons and such you will find the same result. One pattern would be predicted by common descent and another by a common designer but the patterns only fit that of common descent. For example, whales have a complete set of psuedogenes identical to what land based animals possess for their sence of smell. If whales evolved from land based ancestors, this is easily explained. But if they were recently created as is, there is no reason for them to possess such useless genes. A common designer advocate is forced into giving an arbitrary, ad-hoc explanation for this observation.
From here let's move on to other topics. Let's first loook at atavisms. We have brought a few into discussion already. Atavistic legs on whales. Two extra toes on horses. Unfused leg bones in horses. Atavistic tails on humans. The observation is that these atavisms ONLY manifest themselves in a pattern consistent with the phylogenic trees generated from the other lines of evidence. The atavisms only make parts that were possessed by their ancestors in the common descent interpretation. You never see atavisms that fail to follow this pattern. Common descent offers a simple explanation. The common designer option gives no reason why we should expect whales to have genes for making legs of humans to have genes for making tails. They are again forced into capricious explanations.
Development tells a similar story.This has the potential to get rather complicated, so I'll stick with an example already in play. We observe that whales go through a developmental stage in which they possess rear legs. Again, this shared developmental trait follows the same pattern as the other lines of evidence. Common descent offers a simple reason for this to be the case. A common designer has no logical reason to send whales through a stage with legs which must later be reabsorbed.
This can keep going for a long time. If you look at other areas of evidence, you keep coming back to the observation that all the bits always fit the tree that you get from morphology and genetics. This is true for parahomlogy. This is true for vestiges. This is true for the chronology of the fossils. Every observation that you make brings you back to these same trees.
So the question is which interpretation of the data fits the observations. The answer is that common descent offers a simple and compelling answer for each one. A common designer can be hypothesized for some of the observations but for many of the observations, the evidence is the opposite of what would be expected. The only recourse for YEers is to ignore these contradictions. BUt you really must worry about someone's ideas when they must ignore so much.
Take the blinders off and gaze at the following list of beneficial mutations:Originally posted by Gup20:
Darwinian evolution is not simply 'change over time', but an upward change over time. It says that molecules became microbes which became animals which became people. That change has a direction. We don't see that direction in observational science today. We see the opposite direction. We see losses of information, rather than gains.
Mutations finally rendered the genes inoperable. We see MANY instances of this.</font><blockquote>quote:</font><hr />By the way, if you considered the inability of men to make our own vitamin C as a software "bug" in our DNA program, how come it was allowed to get past quality control?
I want to call all the fair minded readers to take note how he has contradicted himself in this statement. How can a scientific textbook be "full of information" and yet you say "it would not be information"?You have mistakenly equated knowlege and information. For example, I can read a large scientific textbook full of information and not understand it. While the information is there... my knowlege may not increase. However, it would not be information UNLESS there was someone or something that could understand it. It would not be information unless it expressed something specific.
Once upon a time, there were no great danes. Now there are. Was this or was this not an increase in information for the genome?What do we see in nature? We see things wearing out... we see things get old and die... we see things decay and dcompose. We don't see an animal born and then increase indefinately... we don't see pepetual motion, etc. We don't see dobermans give birth to spaniels without interbreeding. We do see that a pure doberman mating with a pure doberman gives birth to a pure doberman. No gain in information to suddenly give birth to a great dane, for example, is expected.
A fossil record that you can't prove actually means what you say it means because it was unobserved. The fossils could just as easily be extinc animals created exactly the that way.</font>[/QUOTE]I'm sorry, the ideat that we are not observing fossils is simply ludicrous!</font><blockquote>quote:</font><hr />This is completely contrary to observation of the fossil record, which shows increasing complexity and variation with time until some disaster wipes out a lot of life, then things are simpler again until again increasing complexity is able to be built up.
Yes, and the real geologists tried to do that and it wouldn't work for them and they opened their minds to the alternative views that DO work. However, its nice to see you confirm that, as we already knew, you are absolutely refusing to accept any reinterpreation of what you think you already know, and that no new information has a chance with YOU.Actually it's built upon a straight forward reading of scripture as the basis of our a priori assumptions with which we interpret the scientific data. For example, the Bible says we were created from dust ~ 6000 years ago and that there was a global flood. When we look at the fossil record we say "how could this have come to be in 6000 years?". We look at the rock layers and say "how would a global catastrophe as devastating as flooding the whole earth effect this strata".
The comment remains true in spite of your massive cut and paste that did not address the issue.me, earlier: The flat statement that "information can only come from greator information" is a little strange to be seen on the internet, which would have been totally impossible to create by men just a hundred years ago, due to a total lack of the necessary information as to how to do it.
Lets say you were filling up a bathtub, and it was starting to overflow. Would I need to be able to quantify the rate of overflow to demonstrate that the overflow exists? No. I can take a common sense look at what is happening and tell you - "hey - your tub is overflowing". Determining by how much - or what ammount of water will need to be removed from the floor is the job of a professional.It needs to be if you plan to participate effectively in such discussions. It basically means to show a specific measure of a quantity. This is what is being requested from you and you still fail to provide.
I have provided you with a definition of information. Compare what you have to that definition - if it fits it is information. If it does not fit... then it is not information.If you claim that mutations cannot produce new information then you need to define information in a quantifiable manner such that we can say that this genetic sequence had X amount of information before the mutation and now it has Y amount of information and X is greater then Y.
There is absolutely zero information in either line. Why?Let me know when you can take a sequence such as
atcgatcgctacgctcgatatcgatctagctatcgatc
that then mutates into
atcgatccctacgctcgatatcgatctagctatcgatc
and tell me how much information is in the first sequence and how much information is in the second sequence.
I guess someone would have to have mapped every living organism that has ever lived to know this. Until then, it is just a guess.Two things. First off, genetics is not like a book. Your analogy fails. Mutations would be more akin to creating whole new words. Some are adopted and some are not.
In keeping with our membrane analogy -Second, you have yet to address my answer to Gitt's assertions.
I am keenly aware of what you have been lead to believe. But with the same level of scientific credibility, I could do the following:In addition the example I gave above, let's talk about more.
Whoa there nelly - never did we say that mutations were not beneficial. We are clear to point out that they are in the wrong direction. For example, on windy islands we see winged beetles experience mutations where they loose their wings. This is a beneficial mutation, but is directionally the wrong kind of change for microbes to man evolution. This is evendence of man to microbes evolution as information is LOST (as all the winged beetles are blown out to sea).Take the blinders off and gaze at the following list of beneficial mutations:
There are other possible explainations. For example, they could have a similar cause or reason for the malfunction. For example, lets say some pollutant gets in the water. Lets say this pollutant causes mutations in a specific gene that has receptor cells for the elements in that pollutant. The gene shuts down in all creatures exposed in the same way because the reason for the mutation was common - it attacked specific receptor cells in both man and beast.You've failed to understand that cannot be the case in your vision of the universe because mutations are, in fact, random. The random nature of mutations would force the damaged gene for making vitamin c to be different in different species if they occurred independently. Only if the damaged gene were inherited from the same ancestor would the damage come through as being the same. So why is the damaged gene for all primates the same in species after species? We've allready told you the answer - a common ancestor.
Now lets say the whole class has the same mistakes. Either the whole class has colluded and cheated or perhaps (more likely) the instructor taught wrong or the cirriculum they studied had the wrong answer. Again, we can look to the designer's side as a much more likely source of broad sweeping uniformity.Its like a test paper turned in and you suspect the one copied the other . . . and you take duplicate mistakes as evidence that copying took place. It REALLY IS very good evidence!
In the presence of a decoder to understand the book (aka someone who understands it) it becomes useful infomration. The same is true for our DNA. It can mutate and transform into an oblivion of different things, but unless it means something to the DNA (for example - make a membrane, or make a mitochondria) then it isn't information - it's just code.I want to call all the fair minded readers to take note how he has contradicted himself in this statement. How can a scientific textbook be "full of information" and yet you say "it would not be information"?
How did great danes come to be? Unnatural sexual selection. They were bread that way. What this means is that specific qualities already resident in the genome were isolated and selected.Once upon a time, there were no great danes. Now there are. Was this or was this not an increase in information for the genome?
Are you trying to proove my point or yours? If you tried to breed great danes out of wolves and were unsuccessful, what could you say for sure? That there were no genes for great danes present in wolves. Again, this shows a greater level of diversification in the past, and a higher degree of loss and specific expression today. This is exactly THE OPPOSITE direction of what we should see if evolution is true today. We should see a multitude of animals that could be bread into other species easily as they have all this built up information from evolution. However, this isn't what we see. We see that poodles can no longer have offspring that can be bread into danes - at least not without sexual re-combination. This is precisely what we would predict using the Biblical model of kinds. Clearly, wolves and great danes are members of the dog kind. If you traced it back, you would eventually find a dog from which both great dane and wolf could be selectively bread.Go ahead - mate some wolves and see how many great danes pop out for you.
Doesn't evolution assume that dinosaurs died out 60 million years before man evolved? IF that is true, then how could man have observed live dinosaurs?I'm sorry, the ideat that we are not observing fossils is simply ludicrous!
A nice bit of revisionist history there... but truthfully it was men like Lyell - who it has become known planned out how to remove God from all scientific endevours - who early fostered the ideas of millions of years using geology.Yes, and the real geologists tried to do that and it wouldn't work for them and they opened their minds to the alternative views that DO work.
Act 26:14 And when we were all fallen to the earth, I heard a voice speaking unto me, and saying in the Hebrew tongue, Saul, Saul, why persecutest thou me? [it is] hard for thee to kick against the pricks.However, its nice to see you confirm that, as we already knew, you are absolutely refusing to accept any reinterpreation of what you think you already know, and that no new information has a chance with YOU.
And this, according to evolution, happens by a series of losses and natural selection - a means by which information is discarded.Folks, it is the nature of information to build on itself and increase. This happens to all of us all our lives. It is the reason for the rise and growth of science. It is the reason our hard drives keep filling up in our computers. It is the reason evolution works.
"Until something is specified, it is not information. Moreover until there is a command to the cells to do something they have never done before, it is not new information."But what about mutation? A simple random mutation, while again fulfilling Shannon information, will not meet Gitt's new version of information.
But if that mutation makes a new gene which codes for something useful, things change. We still have the underlying syntax of the genetic code, so the semantic and pragmatic level are undeniably met. But since the mutated version is useful, it now has a purpose and meets the reqirement of the apobetic level as well. Once the gene mutates into something useful, then its purpose cannot be differentiated in any way from the purpose of any other functional gene. The requirements for even Gitt have been met by this process. New information has been created in a manner consistent with evolution's requirements.
"For example, on windy islands we see winged beetles experience mutations where they loose their wings."The question to be asked is what interpretation best explains the evidence that we observe. As new information comes in, it will either support your interpretation or contradict it. Support tells you that you are on the right track. Contradiction tells you that you need to rethink your theory.
The first observation that gets discussed is morphology. Now if we look at the animals that are alive today and the animals that the fossil record tells us were alive in the past, we see that the form a nested heirarchy. All by itself this could mean that all of these animals were produced by common descent or by common designer. (As a note, I am not trying to set up a false dilemma here. I recognize that there may be other explanations that could be put forward but I am purposely restricting the discussion to the two possibilities under discussion.) So you have to go to the next observation.
The next most obvious observation is genetic. If you examine all the different types of genetic material that has been tested and use it to construct phylogenic trees, you find that you get much the same pattern as you do when you arrange the fossils by morphology. Let's see how these observations stack up.
One easy test is to look at just the functional genes. These can again be used to support common descent or a common designer. Both will claim that creatures that are the most similar should have the most similar DNA.
But you can start to untangle the two by looking into further types of genetic material that is not related to the functional part of DNA. One example would be to look at retroviral inserts. These happen when a virus inserts part of its genome into its host. If this happens in a reproductive cell, then the genome of the virus can be passed on to the offspring. Since this has nothing to do with the functional part of the genome, it can shed light on the situation for us. For example, if common descent were true, then you would expect the retroviral DNA to show the same pattern as the other lines of observation. If a common designer were the true explanation, then you would expect a random distribution of the retroviral inserts when compared between the species. In fact, you see that the pattern follws that which wbe expected of common descent. The common designer option cannot explain this pattern.
If you take the retroviral discussion and repeat it with things like paralogs, pseudogenes, retrotransposons and such you will find the same result. One pattern would be predicted by common descent and another by a common designer but the patterns only fit that of common descent. For example, whales have a complete set of psuedogenes identical to what land based animals possess for their sence of smell. If whales evolved from land based ancestors, this is easily explained. But if they were recently created as is, there is no reason for them to possess such useless genes. A common designer advocate is forced into giving an arbitrary, ad-hoc explanation for this observation.
From here let's move on to other topics. Let's first loook at atavisms. We have brought a few into discussion already. Atavistic legs on whales. Two extra toes on horses. Unfused leg bones in horses. Atavistic tails on humans. The observation is that these atavisms ONLY manifest themselves in a pattern consistent with the phylogenic trees generated from the other lines of evidence. The atavisms only make parts that were possessed by their ancestors in the common descent interpretation. You never see atavisms that fail to follow this pattern. Common descent offers a simple explanation. The common designer option gives no reason why we should expect whales to have genes for making legs of humans to have genes for making tails. They are again forced into capricious explanations.
Development tells a similar story.This has the potential to get rather complicated, so I'll stick with an example already in play. We observe that whales go through a developmental stage in which they possess rear legs. Again, this shared developmental trait follows the same pattern as the other lines of evidence. Common descent offers a simple reason for this to be the case. A common designer has no logical reason to send whales through a stage with legs which must later be reabsorbed.
This can keep going for a long time. If you look at other areas of evidence, you keep coming back to the observation that all the bits always fit the tree that you get from morphology and genetics. This is true for parahomlogy. This is true for vestiges. This is true for the chronology of the fossils. Every observation that you make brings you back to these same trees.
So the question is which interpretation of the data fits the observations. The answer is that common descent offers a simple and compelling answer for each one. A common designer can be hypothesized for some of the observations but for many of the observations, the evidence is the opposite of what would be expected. The only recourse for YEers is to ignore these contradictions. BUt you really must worry about someone's ideas when they must ignore so much.
Read the whole thing here.Although natural selection leads to more constrained messages, proteins do not generally use highly patterned or repetitive codon choices. Their amino acid sequences are approximately pseudo-random. The Shannon-Weaver index reaches nearly the maximum value of 2 bits per site for the protein-coding exons of these two Drosophila genes. Regions of repetitive DNA, on the other hand, have low complexity. In the Adh region of the Drosophila genome, these seem to be associated with AT-rich sequences. It is interesting that intron DNA between Adh and outspread exons appears to be organized into sub-regions having different complexities.