You may be through with this thread, but your misinformation needs to be corrected before it infects anyone else. I must divide your posts up so it will take multiple replies.
"But that's okay, neither can Michael Denton. He is an avowed evolutionist, but effectively derailed Darwinism as a viable evolutionary theory in his book 'Evolution: A Theory in Crisis'. He is a more prominent and knowledgable scientist than yourself, and he says that the systems such as the eye or the wing are irreducibly complex and had to have 'suddenly appeared.'"
Your source has lied to you by not being complete. When he wrote the book, he was a young earther. He has since wrtitten another book called Nature's Destiny in which he shows where all he had to say in the first book was wrong. Why don't you read that book if you find Denton to be a credible source.
"And concerning mutations, can you name one occurance of a mutation that was observed to to resulte in an advantage for an organism?
Every mutation ever observed was the result of corrupted DNA info, and has always resulted in weakness or death.
Every one.
Always.
Never has anyone ever seen a genetic mutation occur that gave someone an advantage.
Ever."
Again, your sources must keep you in the dark about the truth in order for you to be able to make such a claim. They are not honest.
But there are ways to make new genes without being harmful. One way is duplication and mutation. The mutation can take many different forms. Here is one example.
"Adaptive evolution after gene duplication," Hughes AL, Trends Genetics, 2002 Sep.18(9):433-4.
A gene was duplicated and one of the copies mutated to give rise to a new function.
In a variation on this theme, two genes that have been duplicated can be combined into a new gene. Here is an example of that.
"Selective sweep of a newly evolved sperm-specific gene in Drosophila," Nurminsky DI, Nurminskaya MV, De Aguiar D, Hartl DL, Nature. 1998 Dec 10;396(6711):572-5.
Another way to produce new proteins exists without even having to mutate the genes. It is called alternative splicing. Genes consists of exon and intron sections. During the process of converting the DNA to protein, the introns are removed and the exons spliced together. Yet organisms can change this splicing such that while the original protein is still made, a new protein is also made by various changes to the process. Maybe an intron is not removed. Maybe an extra exon is removed. There are even more complicated examples. In each case, a new protein is formed from the same raw genetic material without changing the protein that works. When you add point mutations and the copying around of transposable elements, this becomes an even better way to make new proteins.
Another way to get new proteins is to get them from other species. An example. A retrovirus can insert its genetic material into its host. Sometimes this happens in a germ line cell and the virus DNA gets passed on. It happens often enough that a sizable percentage of our DNA are retroviral inserts. These genes can also be subject to mutation and might be made into something useful. Here is an example of that happening.
"Syncytin is a captive retroviral envelope protein involved in human placental morphogenesis," Mi S, Lee X, Li X, Veldman GM, Finnerty H, Racie L, LaVallie E, Tang XY, Edouard P, Howes S, Keith JC Jr, McCoy JM, Nature 2000 Feb 17;403(6771):785-9.
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Let's look specifically at the evolution of resistance to the antibiotic vancomycin.
Vancomycin works by attacking the D-alanyl-D-alanine in the cell wall of the bacterium. There are two genes, VanR and VanS, whose job is to make proteins to detect the presence of vancomycin. When detected, a cascade of other enzymes are created to protect the cell. VanH starts by converting precursor materials into D-lactate. VanA then joins the D-lactate with D-alanyl to make D-alanyl-D-lactate, instead of D-alanyl-D-alanine which is usually used in the cell wall. VanX hydrolyzes the D-alanyl-D-alanine that is still being made before it can be used in the cell wall.
This is the usual process, but there are variations. Some bacteria have VanB instead of VanA to make D-alanyl-D-lactate. Some bacteria replace the D-alanyl instead and make D-serine-D-alanine component instead of D-alanyl-D-lactate.
Once the resistance evolved, it was spread through plasmids.
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Here is a short list of documented cases of beneficial mutations I posted on another thread. A little time with pubmed and you could make a list pages long if you knew how to search effectively.
"Selective sweep of a newly evolved sperm-specific gene in Drosophila," Nurminsky DI, Nurminskaya MV, De Aguiar D, Hartl DL, Nature. 1998 Dec 10;396(6711):572-5.
"Adaptive evolution after gene duplication," Hughes AL, Trends Genetics, 2002 Sep.18(9):433-4.
"Accelerated protein evolution and origins of human-specific features: Foxp2 as an example," Zhang J, Webb DM, Podlaha O, Genetics. 2002 Dec;162(4):1825-35.
"Syncytin is a captive retroviral envelope protein involved in human placental morphogenesis," Mi S, Lee X, Li X, Veldman GM, Finnerty H, Racie L, LaVallie E, Tang XY, Edouard P, Howes S, Keith JC Jr, McCoy JM, Nature 2000 Feb 17;403(6771):785-9.
"Origin of antifreeze protein genes: A cool tale in molecular evolution," John M. Logsdon Jr. and W. Ford Doolittle, Proceedings of the National Academy of Science, USA,Vol. 94, pp. 3485-3487, April 1997.
DeVries, A. L. & Wohlschlag, D. E. (1969) Science 163, 1073-1075.
"A carrot leucine-rich-repeat protein that inhibits ice recrystallization," Worrall D, Elias L, Ashford D, Smallwood M, Sidebottom C, Lillford P, Telford J, Holt C, Bowles D, Science. 1998 Oct 2;282(5386):115-7.
"Recruitment of a double bond isomerase to serve as a reductive dehalogenase during biodegradation of pentachlorophenol," Anandarajah K, Kiefer PM Jr, Donohoe BS, Copley SD, Biochemistry 2000 May 9;39(18):5303-11.
"The Tre2 (USP6) oncogene is a hominoid-specific gene," Paulding CA, Ruvolo M, Haber DA, Proceedings of the National Academy of Science U S A 2003 Mar 4;100(5):2507-11.
"The human genome contains many types of chimeric retrogenes generated through in vivo RNA recombination," Anton Buzdin*, Elena Gogvadze, Elena Kovalskaya, Pavel Volchkov, Svetlana Ustyugova, Anna Illarionova, Alexey Fushan, Tatiana Vinogradova and Eugene Sverdlov, Nucleic Acids Research, 2003, Vol. 31, No. 15 4385-4390.
"The narrow sheath Duplicate Genes: Sectors of Dual Aneuploidy Reveal Ancestrally Conserved Gene Functions During Maize Leaf Development," Michael J. Scanlona, K. David Chenb, and Calvin C. McKnight, IV, Genetics, Vol. 155, 1379-1389, July 2000.
"The maize duplicate genes narrow sheath1 and narrow sheath2 encode a conserved homeobox gene function in a lateral domain of shoot apical meristems," Judith Nardmann1, Jiabing Ji, Wolfgang Werr, and Michael J. Scanlon, Development 131, 2827-2839 (2004).